miR-378a enhances the sensitivity of liver cancer to sorafenib by targeting VEGFR, PDGFRβ and c-Raf

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Publicado en:Molecular Medicine Reports vol. 17, no. 3 (2018), p. 4581
Autor principal: Fu, Hongxia
Otros Autores: Zhang, Jicai, Pan, Tongshan, Ai, Shuying, Tang, Li, Wang, Fengqin
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Spandidos Publications UK Ltd.
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022 |a 1791-2997 
022 |a 1791-3004 
024 7 |a 10.3892/mmr.2018.8390  |2 doi 
035 |a 1994736655 
045 2 |b d20180101  |b d20181231 
100 1 |a Fu, Hongxia 
245 1 |a miR-378a enhances the sensitivity of liver cancer to sorafenib by targeting VEGFR, PDGFRβ and c-Raf 
260 |b Spandidos Publications UK Ltd.  |c 2018 
513 |a Journal Article 
520 3 |a Liver cancer is a globally prevalent cancer with poor prognosis. The present study investigated the link between microRNA-378a (miR-378a) expression and the sensitivity of hepatocellular carcinoma (HCC) and hepatoblastoma (HB) cancers to sorafenib therapy. miR-378a expression was determined in liver tissue samples from healthy candidates and patients with liver cancer using the reverse transcription-quantitative polymerase chain reaction. The antitumor effects of miR-378a alone and in combination with sorafenib were investigated in the HB cell line HepG2 and the HCC cell line SMMC-7721 with methyl thiazoyl tetrazolium, colony formation, flow cytometry and Transwell migration assays. The underlying mechanisms were investigated using western blot analysis. miR-378a expression was decreased in tissue samples from patients with liver cancer. HCC and HB cell line proliferation and invasion ability was inhibited by miR-378a. The combination of miR-378a and sorafenib provided the greatest inhibition. Western blot indicated that mitogen activated protein kinase signaling pathway proteins, vascular endothelial growth factor receptor, platelet derived growth factor receptor β, Raf-1 proto-oncogene, serine/threonine kinase and matrix metallopeptidase 2 were regulated by miR-378a alone and to a greater extent when combined with sorafenib. Results suggest that miR-378a can inhibit liver cancer cell growth and enhance the sensitivity of liver cancer cells to sorafenib-based chemotherapies. 
651 4 |a United States--US 
651 4 |a China 
653 |a Standard deviation 
653 |a Vascular endothelial growth factor 
653 |a Protein-serine/threonine kinase 
653 |a Liver cancer 
653 |a Antitumor activity 
653 |a MicroRNAs 
653 |a Polymerase chain reaction 
653 |a Raf protein 
653 |a Flow cytometry 
653 |a Apoptosis 
653 |a Kinases 
653 |a Proteins 
653 |a Growth factors 
653 |a Medical prognosis 
653 |a Hepatocellular carcinoma 
653 |a Studies 
653 |a Vascular endothelial growth factor receptors 
653 |a Metalloproteinase 
653 |a Hepatocytes 
653 |a Tumors 
653 |a Signal transduction 
653 |a Reverse transcription 
653 |a miRNA 
653 |a Targeted cancer therapy 
653 |a Inhibitor drugs 
700 1 |a Zhang, Jicai 
700 1 |a Pan, Tongshan 
700 1 |a Ai, Shuying 
700 1 |a Tang, Li 
700 1 |a Wang, Fengqin 
773 0 |t Molecular Medicine Reports  |g vol. 17, no. 3 (2018), p. 4581 
786 0 |d ProQuest  |t Health & Medical Collection 
856 4 1 |3 Citation/Abstract  |u https://www.proquest.com/docview/1994736655/abstract/embedded/75I98GEZK8WCJMPQ?source=fedsrch 
856 4 0 |3 Full Text  |u https://www.proquest.com/docview/1994736655/fulltext/embedded/75I98GEZK8WCJMPQ?source=fedsrch 
856 4 0 |3 Full Text - PDF  |u https://www.proquest.com/docview/1994736655/fulltextPDF/embedded/75I98GEZK8WCJMPQ?source=fedsrch