Genetically encoded cell-death indicators (GEDI) to detect an early irreversible commitment to neurodegeneration

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:bioRxiv (Mar 13, 2021), p. n/a
मुख्य लेखक: Linsley, Jeremy W
अन्य लेखक: Shah, Kevan, Castello, Nicholas, Chan, Michelle, Haddad, Dominic, Mancini, Jay, Oza, Viral, Wang, Shijie, Javaherian, Ashkan, Kokel, David, Finkbeiner, Steven
प्रकाशित:
Cold Spring Harbor Laboratory Press
विषय:
ऑनलाइन पहुंच:Citation/Abstract
Full text outside of ProQuest
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LEADER 00000nab a2200000uu 4500
001 2272977154
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022 |a 2692-8205 
024 7 |a 10.1101/726588  |2 doi 
035 |a 2272977154 
045 0 |b d20210313 
100 1 |a Linsley, Jeremy W 
245 1 |a Genetically encoded cell-death indicators (GEDI) to detect an early irreversible commitment to neurodegeneration 
260 |b Cold Spring Harbor Laboratory Press  |c Mar 13, 2021 
513 |a Working Paper 
520 3 |a Abstract Cell death is a critical process that occurs normally in health and disease. However, its study is limited due to available technologies that only detect very late stages in the process or specific death mechanisms. Here, we report the development of a new fluorescent biosensor called genetically encoded death indicator (GEDI). GEDI specifically detects an intracellular Ca2+ level that cells achieve early in the cell death process and marks a stage at which cells are irreversibly committed to die. The time-resolved nature of GEDI delineates a binary demarcation of cell life and death in real time, reformulating the definition of cell death. We demonstrate that GEDI acutely and accurately reports death of rodent and human neurons in vitro, and show GEDI enables a novel automated imaging platform for single cell detection of neuronal death in vivo in zebrafish larvae. With a quantitative pseudo-ratiometric signal, GEDI facilitates high-throughput analysis of cell death in time lapse imaging analysis, providing the necessary resolution and scale to identify early factors leading to cell death in studies of neurodegeneration. Competing Interest Statement The authors have declared no competing interest. 
653 |a Neurodegeneration 
653 |a Biosensors 
653 |a Cell death 
653 |a Calcium (intracellular) 
653 |a Apoptosis 
700 1 |a Shah, Kevan 
700 1 |a Castello, Nicholas 
700 1 |a Chan, Michelle 
700 1 |a Haddad, Dominic 
700 1 |a Mancini, Jay 
700 1 |a Oza, Viral 
700 1 |a Wang, Shijie 
700 1 |a Javaherian, Ashkan 
700 1 |a Kokel, David 
700 1 |a Finkbeiner, Steven 
773 0 |t bioRxiv  |g (Mar 13, 2021), p. n/a 
786 0 |d ProQuest  |t Biological Science Database 
856 4 1 |3 Citation/Abstract  |u https://www.proquest.com/docview/2272977154/abstract/embedded/ZKJTFFSVAI7CB62C?source=fedsrch 
856 4 0 |3 Full text outside of ProQuest  |u https://www.biorxiv.org/content/10.1101/726588v2