Metabolomic profiling reveals grade-specific niacinamide accumulation and its therapeutic Potential via SIRT1-CD38-EMT axis modulation in cervical cancer progression

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Detalles Bibliográficos
Publicado en:bioRxiv (Dec 20, 2024)
Autor principal: Jaiswal, Shivani
Otros Autores: Mishra, Vivek, Majumder, Srija, Wangikar, Pramod P, Sengupta, Shinjinee
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Cold Spring Harbor Laboratory Press
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Acceso en línea:Citation/Abstract
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024 7 |a 10.1101/2024.12.17.628866  |2 doi 
035 |a 3147576523 
045 0 |b d20241220 
100 1 |a Jaiswal, Shivani 
245 1 |a Metabolomic profiling reveals grade-specific niacinamide accumulation and its therapeutic Potential via SIRT1-CD38-EMT axis modulation in cervical cancer progression 
260 |b Cold Spring Harbor Laboratory Press  |c Dec 20, 2024 
513 |a Working Paper 
520 3 |a Despite the availability of new therapies for cervical cancer, innovative strategies are essential to address challenges related to drug resistance and high toxicity. The present study focuses on the metabolic profiling of cervical carcinoma using non-targeted metabolomics approach using liquid chromatography-mass spectrometry. Our study identified over 100 metabolites in cervical tissue samples (both cancerous and adjacent normal) using HILIC and reversed-phase chromatography in the positive and negative ionization modes. The major metabolic alterations included changes in glycolysis, the citric acid cycle, amino acid metabolism, nucleotide metabolism, and nicotinamide metabolism in a grade-dependent manner. Interestingly, metabolic differences between HPV-positive and HPV-negative tumors included elevated arginine and proline metabolism, nicotinamide metabolism, and phosphatidylcholine biosynthesis. We further validated our findings by analyzing transcriptomics datasets from the Gene Expression Omnibus database to understand the expression patterns of the underlying genes involved in the dysregulated pathways. We observed that nicotinamide metabolism exhibits significant effects in lower-grade cervical cancers and specific HPV genotypes. We treated cervical cancer cell lines with niacinamide (NAM), an amide form of niacin, to evaluate its potential therapeutic efficacy. NAM treatment modulated NAD+ metabolism with key players such as CD38, PARP, NAMPT, and SIRT1, promoting apoptosis and inhibiting cell proliferation in cervical cancer cells. Importantly, the metabolic responses differed between the HPV-positive SiHa cells and HPV-negative C33A cells, reflecting distinct NAD+ metabolic adaptations. The study highlights the metabolic adaptation or shifts in cancer progression and provides insights into NAM's molecular mechanisms and therapeutic potential for precision medicine in cervical cancer.Competing Interest StatementThe authors have declared no competing interest.Footnotes* https://www.ebi.ac.uk/metabolights/MTBLS10405 
653 |a Liquid chromatography 
653 |a Precision medicine 
653 |a Cervical carcinoma 
653 |a Amino acid sequence 
653 |a Drug resistance 
653 |a Cell proliferation 
653 |a Toxicity 
653 |a Molecular modelling 
653 |a Phosphatidylcholine 
653 |a Metabolism 
653 |a Transcriptomics 
653 |a Chromatography 
653 |a Genotypes 
653 |a Human papillomavirus 
653 |a Metabolic response 
653 |a CD38 antigen 
653 |a Medical innovations 
653 |a Ionization 
653 |a Cervical cancer 
653 |a Apoptosis 
653 |a Glycolysis 
653 |a Nicotinamide 
653 |a Tumor cell lines 
653 |a Mass spectroscopy 
653 |a Citric acid 
653 |a Gene expression 
653 |a Lecithin 
653 |a Tricarboxylic acid cycle 
653 |a Metabolites 
700 1 |a Mishra, Vivek 
700 1 |a Majumder, Srija 
700 1 |a Wangikar, Pramod P 
700 1 |a Sengupta, Shinjinee 
773 0 |t bioRxiv  |g (Dec 20, 2024) 
786 0 |d ProQuest  |t Biological Science Database 
856 4 1 |3 Citation/Abstract  |u https://www.proquest.com/docview/3147576523/abstract/embedded/6A8EOT78XXH2IG52?source=fedsrch 
856 4 0 |3 Full text outside of ProQuest  |u https://www.biorxiv.org/content/10.1101/2024.12.17.628866v1