Esr1-Dependent Signaling and Transcriptional Maturation in the Medial Preoptic Area of the Hypothalamus Shapes the Development of Mating Behavior during Adolescence

I tiakina i:
Ngā taipitopito rārangi puna kōrero
I whakaputaina i:bioRxiv (Feb 26, 2025)
Kaituhi matua: Hashikawa, Koichi
Ētahi atu kaituhi: Hashikawa, Yoshiko, Briones, Brandy, Ishii, Kentaro K, Liu, Yuejia, Rossi, Mark, Basiri, Marcus L, Chen, Jane Y, Ahmad, Omar, Mukundan, Rishi, Johnston, Nathan, Rhiana Simon, Soetedjo, James, Siputro, Jason, Mchenry, Jenna, Palmiter, Richard D, Rubinow, David, Zweifel, Larry S, Stuber, Garret D
I whakaputaina:
Cold Spring Harbor Laboratory Press
Ngā marau:
Urunga tuihono:Citation/Abstract
Full Text - PDF
Full text outside of ProQuest
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LEADER 00000nab a2200000uu 4500
001 3171518487
003 UK-CbPIL
022 |a 2692-8205 
024 7 |a 10.1101/2025.02.26.640339  |2 doi 
035 |a 3171518487 
045 0 |b d20250226 
100 1 |a Hashikawa, Koichi 
245 1 |a Esr1-Dependent Signaling and Transcriptional Maturation in the Medial Preoptic Area of the Hypothalamus Shapes the Development of Mating Behavior during Adolescence 
260 |b Cold Spring Harbor Laboratory Press  |c Feb 26, 2025 
513 |a Working Paper 
520 3 |a Mating and other behaviors emerge during adolescence through the coordinated actions of steroid hormone signaling throughout the nervous system and periphery. In this study, we investigated the transcriptional dynamics of the medial preoptic area (MPOA), a critical region for reproductive behavior, using single-cell RNA sequencing (scRNAseq) and in situ hybridization techniques in male and female mice throughout adolescence development. Our findings reveal that estrogen receptor 1 (Esr1) plays a pivotal role in the transcriptional maturation of GABAergic neurons within the MPOA during adolescence. Deletion of the estrogen receptor gene, Esr1, in GABAergic neurons (Vgat+) disrupted the developmental progression of mating behaviors in both sexes, while its deletion in glutamatergic neurons (Vglut2+) had no observable effect. In males and females, these neurons displayed distinct transcriptional trajectories, with hormone-dependent gene expression patterns emerging throughout adolescence and regulated by Esr1. Esr1 deletion in MPOA GABAergic neurons, prior to adolescence, arrested adolescent transcriptional progression of these cells and uncovered sex-specific gene-regulatory networks associated with Esr1 signaling. Our results underscore the critical role of Esr1 in orchestrating sex-specific transcriptional dynamics during adolescence, revealing gene regulatory networks implicated in the development of hypothalamic controlled reproductive behaviors.Competing Interest StatementThe authors have declared no competing interest. 
653 |a Child development 
653 |a Hypothalamus 
653 |a Neurons 
653 |a Adolescents 
653 |a Behavior 
653 |a Reproductive behavior 
653 |a Glutamatergic transmission 
653 |a Maturation 
653 |a Gene deletion 
653 |a γ-Aminobutyric acid 
653 |a Hybridization 
653 |a Gene regulation 
653 |a Transcription 
653 |a Preoptic area 
653 |a Gene expression 
653 |a Preoptic area (medial) 
653 |a Nervous system 
653 |a Estrogens 
653 |a Mating behavior 
653 |a Estrogen receptors 
653 |a Adolescence 
653 |a Animal reproduction 
700 1 |a Hashikawa, Yoshiko 
700 1 |a Briones, Brandy 
700 1 |a Ishii, Kentaro K 
700 1 |a Liu, Yuejia 
700 1 |a Rossi, Mark 
700 1 |a Basiri, Marcus L 
700 1 |a Chen, Jane Y 
700 1 |a Ahmad, Omar 
700 1 |a Mukundan, Rishi 
700 1 |a Johnston, Nathan 
700 1 |a Rhiana Simon 
700 1 |a Soetedjo, James 
700 1 |a Siputro, Jason 
700 1 |a Mchenry, Jenna 
700 1 |a Palmiter, Richard D 
700 1 |a Rubinow, David 
700 1 |a Zweifel, Larry S 
700 1 |a Stuber, Garret D 
773 0 |t bioRxiv  |g (Feb 26, 2025) 
786 0 |d ProQuest  |t Biological Science Database 
856 4 1 |3 Citation/Abstract  |u https://www.proquest.com/docview/3171518487/abstract/embedded/7BTGNMKEMPT1V9Z2?source=fedsrch 
856 4 0 |3 Full Text - PDF  |u https://www.proquest.com/docview/3171518487/fulltextPDF/embedded/7BTGNMKEMPT1V9Z2?source=fedsrch 
856 4 0 |3 Full text outside of ProQuest  |u https://www.biorxiv.org/content/10.1101/2025.02.26.640339v1