Connecting developed pressure – Preload relationship in ex-vivo beating heart with cellular sarcomere length – Tension relationship

Đã lưu trong:
Chi tiết về thư mục
Xuất bản năm:Journal of Biomechanics vol. 183 (Apr 2025)
Tác giả chính: Fan, Lei
Tác giả khác: Ziaei-Rad, Vahid, Bazil, Jason, Lee, Lik Chuan
Được phát hành:
Elsevier Limited
Những chủ đề:
Truy cập trực tuyến:Citation/Abstract
Full Text
Full Text - PDF
Các nhãn: Thêm thẻ
Không có thẻ, Là người đầu tiên thẻ bản ghi này!

MARC

LEADER 00000nab a2200000uu 4500
001 3182092875
003 UK-CbPIL
022 |a 0021-9290 
022 |a 1873-2380 
024 7 |a 10.1016/j.jbiomech.2025.112597  |2 doi 
035 |a 3182092875 
045 2 |b d20250422  |b d20250514 
084 |a 170337  |2 nlm 
100 1 |a Fan, Lei 
245 1 |a Connecting developed pressure – Preload relationship in ex-vivo beating heart with cellular sarcomere length – Tension relationship 
260 |b Elsevier Limited  |c Apr 2025 
513 |a Journal Article 
520 3 |a The heart’s developed pressure (DP) in Langendorff heart experiments increases with preload via the Frank-Starling mechanism up to a critical transition point at which DP starts to decrease with preload. A similar behavior is found at the cellular level, where the tension developed by skinned cardiac fibers or myocytes in isometric tension test increases with sarcomere length up to a transition point beyond which, the tension decreases. This cellular-level behavior is termed myofilament length dependent activation. While these two behaviors are similar, they occur at vastly different scales. Specifically, the DP – preload and sarcomere length – tension relationships occur, respectively, at the organ and cellular scales. Correspondingly, it remains unclear how much these behaviors are related. To address this issue, we use computer modeling that connects cellular to organ mechanics found in the ex-vivo beating rat heart experiments to determine whether the DP – preload relationship at the organ level can be explained solely by the sarcomere length – tension relationship at the cellular level. We found that the non-monotonic behavior of the DP with preload is consistent with a model predicted feature of myocardial contractility. The LV sarcomere length at the transition where DP and myocardial contractility start to reduce is 2.12 ± 0.03 μ m. This transition sarcomere length is outside the range of 2.2 – 2.4 μ m that is associated with the peak tension found in skinned rat cardiac fibers or myocytes with isometric tension test. This disparity suggests the presence of other factors affecting the DP – preload relationship found in Langendorff heart experiments such as the prescribed initial length of sarcomere that vary between different rat species. 
653 |a Heart 
653 |a Tension tests 
653 |a Animals 
653 |a Experiments 
653 |a Fibers 
653 |a Myocytes 
653 |a Transition points 
653 |a Ostomy 
653 |a Muscle contraction 
653 |a Computer based modeling 
653 |a Isometric exercise 
653 |a Isometric 
700 1 |a Ziaei-Rad, Vahid 
700 1 |a Bazil, Jason 
700 1 |a Lee, Lik Chuan 
773 0 |t Journal of Biomechanics  |g vol. 183 (Apr 2025) 
786 0 |d ProQuest  |t Health & Medical Collection 
856 4 1 |3 Citation/Abstract  |u https://www.proquest.com/docview/3182092875/abstract/embedded/L8HZQI7Z43R0LA5T?source=fedsrch 
856 4 0 |3 Full Text  |u https://www.proquest.com/docview/3182092875/fulltext/embedded/L8HZQI7Z43R0LA5T?source=fedsrch 
856 4 0 |3 Full Text - PDF  |u https://www.proquest.com/docview/3182092875/fulltextPDF/embedded/L8HZQI7Z43R0LA5T?source=fedsrch