Belt-type electrical muscle stimulation preserves muscle fiber size but does not improve muscle function in a rat model of cancer cachexia

محفوظ في:
التفاصيل البيبلوغرافية
الحاوية / القاعدة:PLoS One vol. 20, no. 11 (Nov 2025), p. e0336391
المؤلف الرئيسي: Kouzaki, Karina
مؤلفون آخرون: Isemura, Mako, Tamura, Yuki, Uno, Hiroyuki, Tadano, Shunta, Akimoto, Ryuji, Hosoki, Katsu, Nakazato, Koichi
منشور في:
Public Library of Science
الموضوعات:
الوصول للمادة أونلاين:Citation/Abstract
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024 7 |a 10.1371/journal.pone.0336391  |2 doi 
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100 1 |a Kouzaki, Karina 
245 1 |a Belt-type electrical muscle stimulation preserves muscle fiber size but does not improve muscle function in a rat model of cancer cachexia 
260 |b Public Library of Science  |c Nov 2025 
513 |a Journal Article 
520 3 |a Cancer cachexia causes severe muscle wasting, and current treatments remain limited. Belt-type electrical muscle stimulation (bEMS) has emerged as a passive rehabilitation tool capable of activating multiple lower limb muscles simultaneously. We investigated whether bEMS prevents muscle wasting and improves functional outcomes in rats with cancer cachexia. Cancer cachexia was induced in male Sprague-Dawley rats by intraperitoneal injection of AH130 Yoshida hepatoma cells. Acute and chronic effects of bEMS were tested. Muscle protein synthesis was evaluated using the SUnSET method, and muscle fiber cross-sectional area (CSA) and ankle torque were measured after chronic stimulation. bEMS increased puromycin-labeled protein levels on day 3 post-injection (~1.5–2.0 fold; p < 0.05). After 10 days, bEMS mitigated reductions in muscle CSA in the gastrocnemius and tibialis anterior compared to the cachexia group. However, muscle strength (ankle torque) was not significantly improved. bEMS preserved muscle fiber size in cancer cachexia but failed to restore muscle function. These findings suggest bEMS may serve as a supportive strategy against muscle atrophy in cachectic conditions. 
610 4 |a Tohoku University 
651 4 |a Japan 
653 |a Ankle 
653 |a Cancer 
653 |a Protein synthesis 
653 |a Hepatoma 
653 |a Recovery of function 
653 |a Animals 
653 |a Chronic illnesses 
653 |a Muscles 
653 |a Protein folding 
653 |a Muscle strength 
653 |a Neuromuscular electrical stimulation 
653 |a Protein biosynthesis 
653 |a Proteins 
653 |a Atrophy 
653 |a Torque 
653 |a Injection 
653 |a Ascites 
653 |a Musculoskeletal system 
653 |a Skeletal muscle 
653 |a Body fat 
653 |a Stimulation 
653 |a Muscle function 
653 |a Puromycin 
653 |a Cachexia 
653 |a Chronic effects 
653 |a Rodents 
653 |a Social 
700 1 |a Isemura, Mako 
700 1 |a Tamura, Yuki 
700 1 |a Uno, Hiroyuki 
700 1 |a Tadano, Shunta 
700 1 |a Akimoto, Ryuji 
700 1 |a Hosoki, Katsu 
700 1 |a Nakazato, Koichi 
773 0 |t PLoS One  |g vol. 20, no. 11 (Nov 2025), p. e0336391 
786 0 |d ProQuest  |t Health & Medical Collection 
856 4 1 |3 Citation/Abstract  |u https://www.proquest.com/docview/3269575021/abstract/embedded/L8HZQI7Z43R0LA5T?source=fedsrch 
856 4 0 |3 Full Text  |u https://www.proquest.com/docview/3269575021/fulltext/embedded/L8HZQI7Z43R0LA5T?source=fedsrch 
856 4 0 |3 Full Text - PDF  |u https://www.proquest.com/docview/3269575021/fulltextPDF/embedded/L8HZQI7Z43R0LA5T?source=fedsrch