Computational Insights into Iron Coordination Disruption in the Human Transferrin–Neisseria meningitidis Bacterial Protein Complex

محفوظ في:
التفاصيل البيبلوغرافية
الحاوية / القاعدة:Inorganics vol. 13, no. 12 (2025), p. 384-411
المؤلف الرئيسي: Dervişoğlu Özdemir Celile
مؤلفون آخرون: Duran, Gizem Nur, Fındık Volkan, Özbil Mehmet, Sağ Erdem Safiye
منشور في:
MDPI AG
الموضوعات:
الوصول للمادة أونلاين:Citation/Abstract
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022 |a 2304-6740 
024 7 |a 10.3390/inorganics13120384  |2 doi 
035 |a 3286306635 
045 2 |b d20250101  |b d20251231 
084 |a 231642  |2 nlm 
100 1 |a Dervişoğlu Özdemir Celile  |u Department of Analytical Chemistry, Faculty of Pharmacy, Istanbul Health and Technology University, Sütlüce Campus, Istanbul 34445, Türkiye; celile.dervisoglu@istun.edu.tr 
245 1 |a Computational Insights into Iron Coordination Disruption in the Human Transferrin–<i>Neisseria meningitidis</i> Bacterial Protein Complex 
260 |b MDPI AG  |c 2025 
513 |a Journal Article 
520 3 |a Among many metal ions in biological systems, iron plays a fundamental role. Transferrins are iron-binding glycoproteins responsible for transporting Fe3+ in vertebrate blood. Neisseria meningitidis, a Gram-negative pathogen causing meningitis, relies on iron for survival and acquires it from human transferrin (hTf) using two surface proteins, TbpA and TbpB. These proteins interact with hTf to form a ternary TbpA–TbpB–hTf complex, enabling iron capture from the host. The absence of an experimental crystal structure for this complex has hindered computational studies, a detailed understanding of Fe3+ dissociation, and designing efficient therapeutics. This study presents the first computational model of the ternary complex, its validation, and molecular dynamics simulations. Structural analyses revealed key electrostatic interactions regulating Fe3+ coordination and essential contact regions between proteins. The role of Lys359 from TbpA was investigated via QM/MM calculations by evaluating Fe3+ binding energies of isolated hTf, the ternary complex, and Lys359Ala, Lys359Arg, Lys359Asp mutant models. Results revealed that the proton transfer from Lys359 leads to disruption of Tyr517–Fe3+ coordination, facilitating iron transfer to the bacterial system. Natural bond orbital analysis confirmed this mechanism. The findings provide new molecular insight into N. meningitidis iron acquisition and identify Lys359 as a potential target for covalent inhibitor design, guiding the development of novel therapeutics against meningococcal infection. 
653 |a Physiology 
653 |a Crystal structure 
653 |a Iron 
653 |a Glycoproteins 
653 |a Protons 
653 |a Ferric ions 
653 |a Meningitis 
653 |a Computer applications 
653 |a Electrostatic properties 
653 |a Transferrins 
653 |a Coordination 
653 |a Drug development 
653 |a Transferrin 
653 |a Drug resistance 
653 |a Proteins 
653 |a Simulation 
653 |a Signal transduction 
653 |a Quantum physics 
653 |a Disruption 
653 |a Ligands 
653 |a Metal ions 
653 |a Molecular dynamics 
653 |a Enzymes 
653 |a Vertebrates 
653 |a Neisseria meningitidis 
700 1 |a Duran, Gizem Nur  |u Institute of Biotechnology, Gebze Technical University, Kocaeli 41400, Türkiye; g.duran2022@gtu.edu.tr 
700 1 |a Fındık Volkan  |u Department of Chemistry, Faculty of Sciences, Marmara University, Göztepe Campus, Istanbul 34722, Türkiye; volkan.findik@marmara.edu.tr 
700 1 |a Özbil Mehmet  |u Institute of Biotechnology, Gebze Technical University, Kocaeli 41400, Türkiye; g.duran2022@gtu.edu.tr 
700 1 |a Sağ Erdem Safiye  |u Department of Chemistry, Faculty of Sciences, Marmara University, Göztepe Campus, Istanbul 34722, Türkiye; volkan.findik@marmara.edu.tr 
773 0 |t Inorganics  |g vol. 13, no. 12 (2025), p. 384-411 
786 0 |d ProQuest  |t Materials Science Database 
856 4 1 |3 Citation/Abstract  |u https://www.proquest.com/docview/3286306635/abstract/embedded/L8HZQI7Z43R0LA5T?source=fedsrch 
856 4 0 |3 Full Text + Graphics  |u https://www.proquest.com/docview/3286306635/fulltextwithgraphics/embedded/L8HZQI7Z43R0LA5T?source=fedsrch 
856 4 0 |3 Full Text - PDF  |u https://www.proquest.com/docview/3286306635/fulltextPDF/embedded/L8HZQI7Z43R0LA5T?source=fedsrch