Exercise training and artery function in humans: nonresponse and its relationship to cardiovascular risk factors

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Bibliografiske detaljer
Udgivet i:Journal of Applied Physiology vol. 117, no. 4 (Aug 15, 2014), p. 345
Hovedforfatter: Green, Daniel J
Andre forfattere: Eijsvogels, Thijs, Bouts, Yvette M, Maiorana, Andrew J, Naylor, Louise H, Scholten, Ralph R, Spaanderman, Marc E A, Pugh, Christopher J A, Sprung, Victoria S, Schreuder, Tim, Jones, Helen, Cable, Tim, Hopman, Maria T E, Thijssen, Dick H J
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American Physiological Society
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100 1 |a Green, Daniel J 
245 1 |a Exercise training and artery function in humans: nonresponse and its relationship to cardiovascular risk factors 
260 |b American Physiological Society  |c Aug 15, 2014 
513 |a Feature 
520 3 |a The objectives of our study were to examine 1) the proportion of responders and nonresponders to exercise training in terms of vascular function; 2) a priori factors related to exercise training-induced changes in conduit artery function, and 3) the contribution of traditional cardiovascular risk factors to exercise-induced changes in artery function. We pooled data from our laboratories involving 182 subjects who underwent supervised, large-muscle group, endurance-type exercise training interventions with pre-/posttraining measures of flow-mediated dilation (FMD%) to assess artery function. All studies adopted an identical FMD protocol (5-min ischemia, distal cuff inflation), contemporary echo-Doppler methodology, and observer-independent automated analysis. Linear regression analysis was used to identify factors contributing to changes in FMD%. We found that cardiopulmonary fitness improved, and weight, body mass index (BMI), cholesterol, and mean arterial pressure (MAP) decreased after training, while FMD% increased in 76% of subjects (P < 0.001). Training-induced increase in FMD% was predicted by lower body weight (β = -0.212), lower baseline FMD% (β = -0.469), lower training frequency (β = -0.256), and longer training duration (β = 0.367) (combined: P < 0.001, r = 0.63). With the exception of a modest correlation with total cholesterol (r = -0.243, P < 0.01), changes in traditional cardiovascular risk factors were not significantly related to changes in FMD% (P > 0.05). In conclusion, we found that, while some subjects do not demonstrate increases following exercise training, improvement in FMD% is present in those with lower pretraining body weight and endothelial function. Moreover, exercise training-induced change in FMD% did not correlate with changes in traditional cardiovascular risk factors, indicating that some cardioprotective effects of exercise training are independent of improvement in risk factors. 
653 |a Exercise 
653 |a Veins & arteries 
653 |a Cardiovascular disease 
653 |a Risk factors 
653 |a Regression analysis 
653 |a Body mass index 
653 |a Cholesterol 
653 |a Physical training 
653 |a Body weight 
653 |a Health risks 
653 |a Social 
700 1 |a Eijsvogels, Thijs 
700 1 |a Bouts, Yvette M 
700 1 |a Maiorana, Andrew J 
700 1 |a Naylor, Louise H 
700 1 |a Scholten, Ralph R 
700 1 |a Spaanderman, Marc E A 
700 1 |a Pugh, Christopher J A 
700 1 |a Sprung, Victoria S 
700 1 |a Schreuder, Tim 
700 1 |a Jones, Helen 
700 1 |a Cable, Tim 
700 1 |a Hopman, Maria T E 
700 1 |a Thijssen, Dick H J 
773 0 |t Journal of Applied Physiology  |g vol. 117, no. 4 (Aug 15, 2014), p. 345 
786 0 |d ProQuest  |t Biological Science Database 
856 4 1 |3 Citation/Abstract  |u https://www.proquest.com/docview/1555410234/abstract/embedded/J7RWLIQ9I3C9JK51?source=fedsrch